| pubmed-article:11499512 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11499512 | lifeskim:mentions | umls-concept:C0026162 | lld:lifeskim |
| pubmed-article:11499512 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:11499512 | lifeskim:mentions | umls-concept:C0011436 | lld:lifeskim |
| pubmed-article:11499512 | lifeskim:mentions | umls-concept:C0332197 | lld:lifeskim |
| pubmed-article:11499512 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:11499512 | pubmed:dateCreated | 2001-8-13 | lld:pubmed |
| pubmed-article:11499512 | pubmed:abstractText | High-resolution synchrotron radiation computed tomography (SRCT) and small-angle x-ray scattering (SAXS) were performed on normal and dentinogenesis imperfecta type II (DI-II) teeth. The SRCT showed that the mineral concentration was 33% lower on average in the DI-II dentin with respect to normal dentin. The SAXS spectra from normal dentin exhibited low-angle diffraction peaks at harmonics of 67.6 nm, consistent with nucleation and growth of the apatite phase within gaps in the collagen fibrils (intrafibrillar mineralization). In contrast, the low-angle peaks were almost non-existent in the DI-II dentin. Crystallite thickness was independent of location in both DI-II and normal dentin, although the crystallites were significantly thicker in DI-II dentin (6.8 nm [SD = 0.5] vs. 5.1 nm [SD = 0.6]). The shape factor of the crystallites, as determined by SAXS, showed a continuous progression in normal dentin from roughly one-dimensional (needle-like) near the pulp to two-dimensional (plate-like) near the dentin-enamel junction. The crystallites in DI-II dentin, on the other hand, remained needle-like throughout. The above observations are consistent with an absence of intrafibrillar mineral in DI-II dentin. | lld:pubmed |
| pubmed-article:11499512 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11499512 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11499512 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11499512 | pubmed:citationSubset | D | lld:pubmed |
| pubmed-article:11499512 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11499512 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11499512 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:11499512 | pubmed:issn | 0022-0345 | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:KinnerJ AJA | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:MarshallG WGW | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:MarshallS JSJ | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:DriessenC HCH | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:BreunigT MTM | lld:pubmed |
| pubmed-article:11499512 | pubmed:author | pubmed-author:PopleJ AJA | lld:pubmed |
| pubmed-article:11499512 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11499512 | pubmed:volume | 80 | lld:pubmed |
| pubmed-article:11499512 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11499512 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11499512 | pubmed:pagination | 1555-9 | lld:pubmed |
| pubmed-article:11499512 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
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| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:meshHeading | pubmed-meshheading:11499512... | lld:pubmed |
| pubmed-article:11499512 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11499512 | pubmed:articleTitle | Intrafibrillar mineral may be absent in dentinogenesis imperfecta type II (DI-II). | lld:pubmed |
| pubmed-article:11499512 | pubmed:affiliation | Department of Preventive and Restorative Dental Sciences, University of California, San Francisco 94143-0758, USA. jkinney@itsa.ucsf.edu | lld:pubmed |
| pubmed-article:11499512 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11499512 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11499512 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11499512 | lld:pubmed |
