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pubmed-article:11470467pubmed:abstractTextMetabolic interventions that promote glucose use during ischemia have been shown to protect the myocardium and improve functional recovery on reperfusion. In this study we evaluated if cardioprotection can be accomplished by inhibiting fatty acid uptake, which would be expected to increase glycolytic metabolism.lld:pubmed
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pubmed-article:11470467pubmed:articleTitleProtection of ischemic hearts perfused with an anion exchange inhibitor, DIDS, is associated with beneficial changes in substrate metabolism.lld:pubmed
pubmed-article:11470467pubmed:affiliationDepartment of Medicine, PH 3-342, Division of Cardiology, College of Physicians and Surgeons, Columbia University, 630 West 168th Street, 10032, New York, NY, USA. rr260@columbia.edulld:pubmed
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