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pubmed-article:11440827pubmed:abstractTextOverproduction of nitric oxide by the inducible form of nitric oxide synthase (iNOS) has been implicated in colitis. Different authors have postulated both toxic and protective effects of nitric oxide (NO) in the pathophysiology of active inflammation. The objective of this study was to examine the role of iNOS in experimental chronic colitis using iNOS-deficient mice.lld:pubmed
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pubmed-article:11440827pubmed:articleTitleReduced sensitivity of inducible nitric oxide synthase-deficient mice to chronic colitis.lld:pubmed
pubmed-article:11440827pubmed:affiliationSecond Department of Internal Medicine, National Defense Medical College, Saitama, Japan.lld:pubmed
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