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pubmed-article:11426325pubmed:abstractTextHuman macrophage inflammatory protein-1 alpha (MIP-1alpha) is a chemotactic cytokine, which binds to macrophages, T cells, and B cells affecting their activation. We found novel polymorphisms at four sites within MIP-1alpha gene in Japanese population: C to T in exon 2; A to G in intron 2; C to G and A to G in exon 3. They occurred on the same allele. Although MIP-1alpha effectively suppresses the replication of HIV-1 in vitro, we observed no statistically significant difference in the allele frequency of this polymorphism between HIV-1-infected and uninfected individuals in Japanese population. Since an increased transcription level of MIP-1alpha has been reported to be associated with inflammatory diseases such as atopic dermatitis, we also investigated the frequency of these polymorphisms among patients with atopic dermatitis, HIV-1-infected individuals (with a normal IgE level), and healthy donors. A small increase in ratio of homozygotes to other genotypes was observed in patients with atopic dermatitis (P = 0.04).lld:pubmed
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pubmed-article:11426325pubmed:dateRevised2007-11-15lld:pubmed
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pubmed-article:11426325pubmed:articleTitleNovel polymorphisms in human macrophage inflammatory protein-1 alpha (MIP-1alpha) gene.lld:pubmed
pubmed-article:11426325pubmed:affiliationDivision of Infectious Diseases, Advanced Clinical Research Center, Institute of Medical Science, University of Tokyo, Tokyo, Japan.lld:pubmed
pubmed-article:11426325pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:11426325pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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