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| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0003591 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0023776 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C1533698 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0441994 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0314603 | lld:lifeskim |
| pubmed-article:11425775 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:11425775 | pubmed:issue | 25 | lld:pubmed |
| pubmed-article:11425775 | pubmed:dateCreated | 2001-6-26 | lld:pubmed |
| pubmed-article:11425775 | pubmed:abstractText | Inducible nitric oxide synthase (iNOS) is expressed by leukocytes and smooth muscle cells in atherosclerotic lesions. To test whether NO produced by iNOS deficiency affects atherosclerosis, we studied apoE/iNOS-double knockout (dKO) and apoE-knockout (KO) control animals fed a "Western-type" diet. | lld:pubmed |
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| pubmed-article:11425775 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11425775 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:11425775 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11425775 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:11425775 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:11425775 | pubmed:author | pubmed-author:ChenJJ | lld:pubmed |
| pubmed-article:11425775 | pubmed:author | pubmed-author:HuangP LPL | lld:pubmed |
| pubmed-article:11425775 | pubmed:author | pubmed-author:HamGG | lld:pubmed |
| pubmed-article:11425775 | pubmed:author | pubmed-author:KuhlencordtP... | lld:pubmed |
| pubmed-article:11425775 | pubmed:author | pubmed-author:AsternJJ | lld:pubmed |
| pubmed-article:11425775 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:11425775 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:11425775 | pubmed:volume | 103 | lld:pubmed |
| pubmed-article:11425775 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11425775 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11425775 | pubmed:pagination | 3099-104 | lld:pubmed |
| pubmed-article:11425775 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:11425775 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11425775 | pubmed:articleTitle | Genetic deficiency of inducible nitric oxide synthase reduces atherosclerosis and lowers plasma lipid peroxides in apolipoprotein E-knockout mice. | lld:pubmed |
| pubmed-article:11425775 | pubmed:affiliation | Cardiovascular Research Center and Cardiology Division, Massachusetts General Hospital, and Harvard Medical School, Boston, Massachusetts, USA. | lld:pubmed |
| pubmed-article:11425775 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11425775 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11425775 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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