| pubmed-article:11381188 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11381188 | lifeskim:mentions | umls-concept:C0600466 | lld:lifeskim |
| pubmed-article:11381188 | lifeskim:mentions | umls-concept:C0026769 | lld:lifeskim |
| pubmed-article:11381188 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
| pubmed-article:11381188 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:11381188 | lifeskim:mentions | umls-concept:C1515021 | lld:lifeskim |
| pubmed-article:11381188 | pubmed:issue | 3-4 | lld:pubmed |
| pubmed-article:11381188 | pubmed:dateCreated | 2001-5-30 | lld:pubmed |
| pubmed-article:11381188 | pubmed:abstractText | In the present study we determined the frequencies of four haplotypes of the human T-cell lymphotropic virus-related endogenous sequence, HRES-1, in 110 multiple sclerosis (MS) patients and 100 healthy control subjects from the United Kingdom. We found evidence of an association between this endogenous retrovirus and MS (p < 0.01), in particular reflecting an increased frequency of HRES-1 haplotype 1 in the group of patients. There was no significant difference in the distribution of HRES-1 haplotypes between relapsing-remitting MS and the primary progressive form of the disease. The odds ratio for HRES-1 haplotype 1 and MS did not differ significantly between individuals positive for HLA-DR2 and DR2-negative individuals. Comparison of the observations from the present study with previous results implicated HRES-1 as a marker of genetic heterogeneity in MS. | lld:pubmed |
| pubmed-article:11381188 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11381188 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11381188 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11381188 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11381188 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11381188 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11381188 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11381188 | pubmed:issn | 0278-0240 | lld:pubmed |
| pubmed-article:11381188 | pubmed:author | pubmed-author:ClausenJJ | lld:pubmed |
| pubmed-article:11381188 | pubmed:author | pubmed-author:KellyM AMA | lld:pubmed |
| pubmed-article:11381188 | pubmed:author | pubmed-author:FrancisD ADA | lld:pubmed |
| pubmed-article:11381188 | pubmed:author | pubmed-author:RasmussenH... | lld:pubmed |
| pubmed-article:11381188 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11381188 | pubmed:volume | 16 | lld:pubmed |
| pubmed-article:11381188 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11381188 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11381188 | pubmed:pagination | 101-4 | lld:pubmed |
| pubmed-article:11381188 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:meshHeading | pubmed-meshheading:11381188... | lld:pubmed |
| pubmed-article:11381188 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11381188 | pubmed:articleTitle | Association between the endogenous retrovirus HRES-1 and multiple sclerosis in the United Kingdom--evidence of genetically different disease subsets? | lld:pubmed |
| pubmed-article:11381188 | pubmed:affiliation | Department of Life Sciences and Chemistry, Roskilde University, PO Box 260, DK-4000 Roskilde, Denmark. | lld:pubmed |
| pubmed-article:11381188 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11381188 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
