| pubmed-article:11328912 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C0019868 | lld:lifeskim |
| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C0521942 | lld:lifeskim |
| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C2347899 | lld:lifeskim |
| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C0332293 | lld:lifeskim |
| pubmed-article:11328912 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
| pubmed-article:11328912 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:11328912 | pubmed:dateCreated | 2001-4-30 | lld:pubmed |
| pubmed-article:11328912 | pubmed:abstractText | The angiotensin II (AT II) type I receptor antagonist losartan has been reported to increase urinary uric acid and potassium excretion. These effects might be beneficial in cyclosporin (CsA)-treated renal transplant recipients, who frequently suffer from hyperuricaemia and hyperkalaemia. | lld:pubmed |
| pubmed-article:11328912 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11328912 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11328912 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11328912 | pubmed:month | May | lld:pubmed |
| pubmed-article:11328912 | pubmed:issn | 0931-0509 | lld:pubmed |
| pubmed-article:11328912 | pubmed:author | pubmed-author:SchmidtAA | lld:pubmed |
| pubmed-article:11328912 | pubmed:author | pubmed-author:MayerGG | lld:pubmed |
| pubmed-article:11328912 | pubmed:author | pubmed-author:BöhmigGG | lld:pubmed |
| pubmed-article:11328912 | pubmed:author | pubmed-author:GruberUU | lld:pubmed |
| pubmed-article:11328912 | pubmed:author | pubmed-author:KöllerEE | lld:pubmed |
| pubmed-article:11328912 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11328912 | pubmed:volume | 16 | lld:pubmed |
| pubmed-article:11328912 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11328912 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11328912 | pubmed:pagination | 1034-7 | lld:pubmed |
| pubmed-article:11328912 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:11328912 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11328912 | pubmed:articleTitle | The effect of ACE inhibitor and angiotensin II receptor antagonist therapy on serum uric acid levels and potassium homeostasis in hypertensive renal transplant recipients treated with CsA. | lld:pubmed |
| pubmed-article:11328912 | pubmed:affiliation | Division of Nephrology and Dialysis, Department of Medicine III, University of Vienna, Währinger Gürtel 18-20, A-1090 Vienna, Austria. | lld:pubmed |
| pubmed-article:11328912 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11328912 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:11328912 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:11328912 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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