Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:11208998rdf:typepubmed:Citationlld:pubmed
pubmed-article:11208998lifeskim:mentionsumls-concept:C0030705lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0006675lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0439849lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0014792lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0085612lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0439793lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C0445223lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C1552599lld:lifeskim
pubmed-article:11208998lifeskim:mentionsumls-concept:C1704787lld:lifeskim
pubmed-article:11208998pubmed:issue1lld:pubmed
pubmed-article:11208998pubmed:dateCreated2001-3-14lld:pubmed
pubmed-article:11208998pubmed:abstractTextMyocardial disorders are a remarkable cause of morbidity and mortality in chronic haemodialysed patients (HD). They could be favoured by alteration of cell Ca(2+) handling. In previous studies we characterized an erythrocyte Ca(2+) influx, sensitive to membrane potential and inhibited by Ca(2+) antagonists. Since its maximal influx rate was decreased in HD patients, this study investigates if Ca(2+) influx alterations are related to myocardial disorders in HD patients.lld:pubmed
pubmed-article:11208998pubmed:languageenglld:pubmed
pubmed-article:11208998pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11208998pubmed:citationSubsetIMlld:pubmed
pubmed-article:11208998pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11208998pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11208998pubmed:statusMEDLINElld:pubmed
pubmed-article:11208998pubmed:monthJanlld:pubmed
pubmed-article:11208998pubmed:issn0931-0509lld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:SpottiDDlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:BianchiGGlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:CusiDDlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:SoldatiLLlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:MelandriMMlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:ManuntaPPlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:StellaPPlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:VezzoliGGlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:SlavieroGGlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:AdamoDDlld:pubmed
pubmed-article:11208998pubmed:authorpubmed-author:CiurlinoDDlld:pubmed
pubmed-article:11208998pubmed:issnTypePrintlld:pubmed
pubmed-article:11208998pubmed:volume16lld:pubmed
pubmed-article:11208998pubmed:ownerNLMlld:pubmed
pubmed-article:11208998pubmed:authorsCompleteYlld:pubmed
pubmed-article:11208998pubmed:pagination85-90lld:pubmed
pubmed-article:11208998pubmed:dateRevised2007-11-15lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:meshHeadingpubmed-meshheading:11208998...lld:pubmed
pubmed-article:11208998pubmed:year2001lld:pubmed
pubmed-article:11208998pubmed:articleTitleErythrocyte calcium influx is related to severity of ventricular arrhythmias in uraemic patients.lld:pubmed
pubmed-article:11208998pubmed:affiliationDivision of Nephrology, Dialysis and Hypertension, San Raffaele Scientific Institute, University of Milan, Italy.lld:pubmed
pubmed-article:11208998pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:11208998pubmed:publicationTypeIn Vitrolld:pubmed
pubmed-article:11208998pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed