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pubmed-article:11180995pubmed:abstractTextBilirubin oxidative metabolites (BOMs) are generated from bilirubin as a result of its scavenging action against free radicals. During sepsis, excess amounts of free radicals are produced, and they play an important role in the pathophysiological process. We studied whether urinary excretion of BOMs would increase under septic conditions in humans and compared BOM levels with other well-established clinical parameters of inflammation.lld:pubmed
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pubmed-article:11180995pubmed:authorpubmed-author:YamaguchiKKlld:pubmed
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pubmed-article:11180995pubmed:copyrightInfoCopyright 2001 Academic Press.lld:pubmed
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pubmed-article:11180995pubmed:volume96lld:pubmed
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pubmed-article:11180995pubmed:articleTitleIncreased urinary excretion of bilirubin oxidative metabolites in septic patients: a new marker for oxidative stress in vivo.lld:pubmed
pubmed-article:11180995pubmed:affiliationDepartment of Surgery and Oncology, Graduate School of Medical Sciences, Kyushu University, Fukuoka, 812-8582, Japan.lld:pubmed
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pubmed-article:11180995pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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