| pubmed-article:11169051 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C1456535 | lld:lifeskim |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C0043047 | lld:lifeskim |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:11169051 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:11169051 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:11169051 | pubmed:dateCreated | 2001-2-22 | lld:pubmed |
| pubmed-article:11169051 | pubmed:abstractText | The influence of a second lethal stress (SLS) was investigated when populations of Escherichia coli M23 OR.H- were exposed to either a low water activity (aw) of 0.90 or a pH of 3.50 after 24 h at 25 degrees C. Regardless of the initial stress, E. coli M23 OR.H- populations initially demonstrated biphasic inactivation kinetics consisting of a rapid first phase of death followed by a slower second phase. When cultures initially exposed to aw 0.90 experienced an SLS of pH 3.50, a second rapid inactivation period was observed before a subpopulation of more resistant cells emerged. This subpopulation was able to persist for approximately 50 h after imposition of the SLS. In contrast, E. coli M23 OR.H- cells first exposed to a pH of 3.50 were inactivated rapidly to levels below the limits of detection upon imposition of an SLS of aw 0.90. It is hypothesized that pH stress constitutes a large energy drain on the cell and subsequently sensitizes it to other environmental constraints requiring expenditure of metabolic energy. | lld:pubmed |
| pubmed-article:11169051 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11169051 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11169051 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11169051 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11169051 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11169051 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11169051 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:11169051 | pubmed:issn | 0266-8254 | lld:pubmed |
| pubmed-article:11169051 | pubmed:author | pubmed-author:RoseFF | lld:pubmed |
| pubmed-article:11169051 | pubmed:author | pubmed-author:McMeekinT ATA | lld:pubmed |
| pubmed-article:11169051 | pubmed:author | pubmed-author:ShadboltCC | lld:pubmed |
| pubmed-article:11169051 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11169051 | pubmed:volume | 32 | lld:pubmed |
| pubmed-article:11169051 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11169051 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11169051 | pubmed:pagination | 99-102 | lld:pubmed |
| pubmed-article:11169051 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:meshHeading | pubmed-meshheading:11169051... | lld:pubmed |
| pubmed-article:11169051 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11169051 | pubmed:articleTitle | Differentiation of the effects of lethal pH and water activity: food safety implications. | lld:pubmed |
| pubmed-article:11169051 | pubmed:affiliation | School of Agricultural Science and Tasmanian Institute of Agricultural Research, University of Tasmania, Hobart, Tasmania, Australia. craig_ts@utas.edu.au | lld:pubmed |
| pubmed-article:11169051 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11169051 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11169051 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11169051 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11169051 | lld:pubmed |
