pubmed-article:11167806 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11167806 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:11167806 | lifeskim:mentions | umls-concept:C0023473 | lld:lifeskim |
pubmed-article:11167806 | lifeskim:mentions | umls-concept:C0178666 | lld:lifeskim |
pubmed-article:11167806 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:11167806 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:11167806 | pubmed:dateCreated | 2001-2-22 | lld:pubmed |
pubmed-article:11167806 | pubmed:abstractText | The regulation of nutrient transport by both cytokines and oncogenes has been linked to haemopoietic cell survival. In this study, we found that activation of Bcr--Abl protein tyrosine kinase was associated with the stimulation of glucose transport in the multipotent haemopoietic cell line FDCP-mix, a cell model for chronic-phase chronic myeloid leukaemia (CML). Bcr--Abl upregulation of glucose transport was mediated by phosphatidylinositol-3-kinase. The observation that Bcr--Abl can regulate glucose transport in a CML cell model raises the possibility that glucose transport regulation may have a role to play in the aberrant survival of stem cells in the chronic phase of CML. | lld:pubmed |
pubmed-article:11167806 | pubmed:language | eng | lld:pubmed |
pubmed-article:11167806 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11167806 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:11167806 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11167806 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11167806 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11167806 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11167806 | pubmed:month | Jan | lld:pubmed |
pubmed-article:11167806 | pubmed:issn | 0007-1048 | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:BentleyJJ | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:WalkerII | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:BaldwinS ASA | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:WhettonA DAD | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:McIntoshEE | lld:pubmed |
pubmed-article:11167806 | pubmed:author | pubmed-author:Owen-LynchP... | lld:pubmed |
pubmed-article:11167806 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11167806 | pubmed:volume | 112 | lld:pubmed |
pubmed-article:11167806 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11167806 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11167806 | pubmed:pagination | 212-5 | lld:pubmed |
pubmed-article:11167806 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
pubmed-article:11167806 | pubmed:meshHeading | pubmed-meshheading:11167806... | lld:pubmed |
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pubmed-article:11167806 | pubmed:meshHeading | pubmed-meshheading:11167806... | lld:pubmed |
pubmed-article:11167806 | pubmed:year | 2001 | lld:pubmed |
pubmed-article:11167806 | pubmed:articleTitle | Glucose transport regulation by p210 Bcr-Abl in a chronic myeloid leukaemia model. | lld:pubmed |
pubmed-article:11167806 | pubmed:affiliation | School of Biochemistry and Molecular Biology, University of Leeds, Leeds, UK. Johanne.Bentley@ncl.ac.uk | lld:pubmed |
pubmed-article:11167806 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11167806 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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