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pubmed-article:11114845pubmed:abstractTextThe autoimmune thyroid diseases (AITDs), comprising Graves' disease (GD) and Hashimoto's thyroiditis (HT), appear to develop as a result of complex interactions between predisposing genes and environmental triggers. Susceptibility to AITDs is conferred by genes in the human leukocyte antigen (HLA) and genes unlinked to HLA, including the CTLA-4 gene. Recently, an association to some estrogen receptor (ER)alpha genotypes with breast cancer, hypertension, osteoporosis, generalized osteoarthritis, and some autoimmune diseases such as rheumatoid arthritis has been reported. We have analyzed a dinucleotide (TA)n repeat polymorphism lying upstream of the human ERalpha gene in patients with AITDs and in normal subjects.lld:pubmed
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pubmed-article:11114845pubmed:languageenglld:pubmed
pubmed-article:11114845pubmed:statusPubMed-not-MEDLINElld:pubmed
pubmed-article:11114845pubmed:authorpubmed-author:BanYYlld:pubmed
pubmed-article:11114845pubmed:authorpubmed-author:TomitaMMlld:pubmed
pubmed-article:11114845pubmed:authorpubmed-author:TaniyamaMMlld:pubmed
pubmed-article:11114845pubmed:authorpubmed-author:TozakiTTlld:pubmed
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pubmed-article:11114845pubmed:pagination1lld:pubmed
pubmed-article:11114845pubmed:dateRevised2009-11-18lld:pubmed
pubmed-article:11114845pubmed:articleTitleEstrogen receptor alpha dinucleotide repeat polymorphism in Japanese patients with autoimmune thyroid diseases.lld:pubmed
pubmed-article:11114845pubmed:affiliationThird Department of Internal Medicine, Showa University School of Medicine, Tokyo, Japan. yshsban@ns2.cc.showa-u.ac.jplld:pubmed
pubmed-article:11114845pubmed:publicationTypeJournal Articlelld:pubmed
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