| pubmed-article:11048727 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11048727 | lifeskim:mentions | umls-concept:C0005477 | lld:lifeskim |
| pubmed-article:11048727 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:11048727 | pubmed:issue | 6805 | lld:pubmed |
| pubmed-article:11048727 | pubmed:dateCreated | 2000-10-25 | lld:pubmed |
| pubmed-article:11048727 | pubmed:abstractText | Apoptosis--the regulated destruction of a cell--is a complicated process. The decision to die cannot be taken lightly, and the activity of many genes influence a cell's likelihood of activating its self-destruction programme. Once the decision is taken, proper execution of the apoptotic programme requires the coordinated activation and execution of multiple subprogrammes. Here I review the basic components of the death machinery, describe how they interact to regulate apoptosis in a coordinated manner, and discuss the main pathways that are used to activate cell death. | lld:pubmed |
| pubmed-article:11048727 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11048727 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11048727 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11048727 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11048727 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11048727 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11048727 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:11048727 | pubmed:issn | 0028-0836 | lld:pubmed |
| pubmed-article:11048727 | pubmed:author | pubmed-author:HengartnerM... | lld:pubmed |
| pubmed-article:11048727 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11048727 | pubmed:day | 12 | lld:pubmed |
| pubmed-article:11048727 | pubmed:volume | 407 | lld:pubmed |
| pubmed-article:11048727 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11048727 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11048727 | pubmed:pagination | 770-6 | lld:pubmed |
| pubmed-article:11048727 | pubmed:dateRevised | 2005-11-16 | lld:pubmed |
| pubmed-article:11048727 | pubmed:meshHeading | pubmed-meshheading:11048727... | lld:pubmed |
| pubmed-article:11048727 | pubmed:meshHeading | pubmed-meshheading:11048727... | lld:pubmed |
| pubmed-article:11048727 | pubmed:meshHeading | pubmed-meshheading:11048727... | lld:pubmed |
| pubmed-article:11048727 | pubmed:meshHeading | pubmed-meshheading:11048727... | lld:pubmed |
| pubmed-article:11048727 | pubmed:meshHeading | pubmed-meshheading:11048727... | lld:pubmed |
| pubmed-article:11048727 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11048727 | pubmed:articleTitle | The biochemistry of apoptosis. | lld:pubmed |
| pubmed-article:11048727 | pubmed:affiliation | Cold Spring Harbor Laboratory, New York 11724, USA. hengartn@cshl.org | lld:pubmed |
| pubmed-article:11048727 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11048727 | pubmed:publicationType | Review | lld:pubmed |
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