pubmed-article:1104749 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:1104749 | lifeskim:mentions | umls-concept:C0012655 | lld:lifeskim |
pubmed-article:1104749 | lifeskim:mentions | umls-concept:C0002726 | lld:lifeskim |
pubmed-article:1104749 | lifeskim:mentions | umls-concept:C0017263 | lld:lifeskim |
pubmed-article:1104749 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
pubmed-article:1104749 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:1104749 | pubmed:dateCreated | 1976-2-26 | lld:pubmed |
pubmed-article:1104749 | pubmed:abstractText | Experimental amyloidosis in mice can be induced by repeated injections of casein. It has now been demonstrated that casein induces strong polyclonal antibody synthesis in mouse B spleen lymphocytes. This effect is much more pronounced in spleen cells from anyloid-susceptible mice (CBA/J) than amyloid-resistant mice (A/J). It is suggested that amyloidosis can be due in some instances to a constant exposure for molecules which induce polyclonal B-cell activation. | lld:pubmed |
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pubmed-article:1104749 | pubmed:language | eng | lld:pubmed |
pubmed-article:1104749 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1104749 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:1104749 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:1104749 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:1104749 | pubmed:month | Dec | lld:pubmed |
pubmed-article:1104749 | pubmed:issn | 0022-1007 | lld:pubmed |
pubmed-article:1104749 | pubmed:author | pubmed-author:BrittonSS | lld:pubmed |
pubmed-article:1104749 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:1104749 | pubmed:day | 1 | lld:pubmed |
pubmed-article:1104749 | pubmed:volume | 142 | lld:pubmed |
pubmed-article:1104749 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:1104749 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:1104749 | pubmed:pagination | 1564-9 | lld:pubmed |
pubmed-article:1104749 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:1104749 | pubmed:year | 1975 | lld:pubmed |
pubmed-article:1104749 | pubmed:articleTitle | Experimental amyloidosis: the inducer is a polyclonal B-cell activator to which susceptibility is under genetic control. | lld:pubmed |
pubmed-article:1104749 | pubmed:publicationType | Journal Article | lld:pubmed |
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