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pubmed-article:11038295pubmed:abstractTextColorectal cancer remains one of the most prevalent malignancies in the United States. Improvement in local disease control is seen when 5-fluorouracil (5-FU) is used in combination with pelvic irradiation for rectal adenocarcinoma. The frequent overexpression of insulin-like growth factor I receptor (IGF-I-R) in rectal adenocarcinoma suggests that inhibition of the signal transduction pathway may be a novel approach to enhance tumor response. This investigation seeks to define the role of IGF-I-R antagonism, using monoclonal antibody alpha-IR3, in augmenting cytotoxicity to adjuvant chemoradiation therapy for adenocarcinoma of the rectum.lld:pubmed
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pubmed-article:11038295pubmed:copyrightInfoCopyright 2000 Academic Press.lld:pubmed
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pubmed-article:11038295pubmed:dateRevised2009-11-19lld:pubmed
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pubmed-article:11038295pubmed:articleTitleInsulin-like growth factor I receptor antagonism augments response to chemoradiation therapy in colon cancer cells.lld:pubmed
pubmed-article:11038295pubmed:affiliationDepartment of Surgery, Tulane University School of Medicine, New Orleans, Louisiana 70118, USA.lld:pubmed
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