pubmed-article:11022084 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0038454 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0019010 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C0475224 | lld:lifeskim |
pubmed-article:11022084 | lifeskim:mentions | umls-concept:C1883709 | lld:lifeskim |
pubmed-article:11022084 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:11022084 | pubmed:dateCreated | 2000-10-13 | lld:pubmed |
pubmed-article:11022084 | pubmed:abstractText | Patients with severe carotid artery stenosis may have more severe ischemic damage after embolic stroke than patients without this abnormality. Unilateral proximal carotid occlusion (UCO) alone typically does not induce infarction in normotensive rats. The aim of this study was to investigate whether UCO increases infarct size after microembolic, experimental stroke. | lld:pubmed |
pubmed-article:11022084 | pubmed:language | eng | lld:pubmed |
pubmed-article:11022084 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11022084 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:11022084 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11022084 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11022084 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11022084 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11022084 | pubmed:month | Oct | lld:pubmed |
pubmed-article:11022084 | pubmed:issn | 1524-4628 | lld:pubmed |
pubmed-article:11022084 | pubmed:author | pubmed-author:POCHRR | lld:pubmed |
pubmed-article:11022084 | pubmed:author | pubmed-author:FisherMM | lld:pubmed |
pubmed-article:11022084 | pubmed:author | pubmed-author:LiFF | lld:pubmed |
pubmed-article:11022084 | pubmed:author | pubmed-author:SotakC HCH | lld:pubmed |
pubmed-article:11022084 | pubmed:author | pubmed-author:Mayzel-OregOO | lld:pubmed |
pubmed-article:11022084 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:11022084 | pubmed:volume | 31 | lld:pubmed |
pubmed-article:11022084 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11022084 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11022084 | pubmed:pagination | 2494-9 | lld:pubmed |
pubmed-article:11022084 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
pubmed-article:11022084 | pubmed:meshHeading | pubmed-meshheading:11022084... | lld:pubmed |
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pubmed-article:11022084 | pubmed:meshHeading | pubmed-meshheading:11022084... | lld:pubmed |
pubmed-article:11022084 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:11022084 | pubmed:articleTitle | Inapparent hemodynamic insufficiency exacerbates ischemic damage in a rat microembolic stroke model. | lld:pubmed |
pubmed-article:11022084 | pubmed:affiliation | Department of Neurology, University of Massachusetts Memorial Health Care and University of Massachusetts Medical School, Worcester, USA. | lld:pubmed |
pubmed-article:11022084 | pubmed:publicationType | Journal Article | lld:pubmed |
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