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pubmed-article:11003799rdf:typepubmed:Citationlld:pubmed
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pubmed-article:11003799pubmed:dateCreated2001-1-26lld:pubmed
pubmed-article:11003799pubmed:abstractTextCiguatera poisoning causes mainly gastrointestinal and neurological effects of variable severity. However, symptoms of peripheral neuropathy with paresthesias and paradoxical disturbance of thermal sensation are the hallmark. Electrophysiological studies are often normal, except in severe cases. We report four people who developed mild ciguatera poisoning after barracuda ingestion. Electrophysiological studies documented normocalcemic latent tetany. These findings are consistent with ciguatoxin's mechanism of toxicity, which involves inactivation of voltage-gated Na(+) channels and eventually increases nerve membrane excitability.lld:pubmed
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pubmed-article:11003799pubmed:authorpubmed-author:ManziJJlld:pubmed
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pubmed-article:11003799pubmed:authorpubmed-author:GandiniCClld:pubmed
pubmed-article:11003799pubmed:authorpubmed-author:LocatelliCClld:pubmed
pubmed-article:11003799pubmed:authorpubmed-author:ButeraRRlld:pubmed
pubmed-article:11003799pubmed:copyrightInfoCopyright 2000 John Wiley & Sons, Inc.lld:pubmed
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pubmed-article:11003799pubmed:volume23lld:pubmed
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pubmed-article:11003799pubmed:pagination1598-603lld:pubmed
pubmed-article:11003799pubmed:dateRevised2004-11-17lld:pubmed
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pubmed-article:11003799pubmed:articleTitleMild ciguatera poisoning: Case reports with neurophysiological evaluations.lld:pubmed
pubmed-article:11003799pubmed:affiliationToxicology Unit, IRCCS Maugeri Foundation Medical Center, Via Ferrata, 8, 27100 Pavia, Italy. rbutera@fsm.itlld:pubmed
pubmed-article:11003799pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:11003799pubmed:publicationTypeCase Reportslld:pubmed