pubmed-article:10982767 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10982767 | lifeskim:mentions | umls-concept:C0009319 | lld:lifeskim |
pubmed-article:10982767 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:10982767 | lifeskim:mentions | umls-concept:C0079189 | lld:lifeskim |
pubmed-article:10982767 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
pubmed-article:10982767 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
pubmed-article:10982767 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:10982767 | pubmed:dateCreated | 2000-9-29 | lld:pubmed |
pubmed-article:10982767 | pubmed:abstractText | Most experimental models for inflammatory bowel disease in mice are associated with production of interferon (IFN)-gamma and other proinflammatory cytokines. We hypothesized that T-helper 2 (Th2)-type cells could also contribute to the colitis and cause inflammation different than that mediated by Th1-type cells. | lld:pubmed |
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pubmed-article:10982767 | pubmed:language | eng | lld:pubmed |
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pubmed-article:10982767 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:10982767 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10982767 | pubmed:month | Sep | lld:pubmed |
pubmed-article:10982767 | pubmed:issn | 0016-5085 | lld:pubmed |
pubmed-article:10982767 | pubmed:author | pubmed-author:ElsonC OCO | lld:pubmed |
pubmed-article:10982767 | pubmed:author | pubmed-author:DohiTT | lld:pubmed |
pubmed-article:10982767 | pubmed:author | pubmed-author:McGheeJ RJR | lld:pubmed |
pubmed-article:10982767 | pubmed:author | pubmed-author:KiyonoHH | lld:pubmed |
pubmed-article:10982767 | pubmed:author | pubmed-author:FujihashiKK | lld:pubmed |
pubmed-article:10982767 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10982767 | pubmed:volume | 119 | lld:pubmed |
pubmed-article:10982767 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10982767 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10982767 | pubmed:pagination | 724-33 | lld:pubmed |
pubmed-article:10982767 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:10982767 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:10982767 | pubmed:articleTitle | Mice deficient in Th1- and Th2-type cytokines develop distinct forms of hapten-induced colitis. | lld:pubmed |
pubmed-article:10982767 | pubmed:affiliation | Immunobiology Vaccine Center, University of Alabama at Birmingham, Birmingham, Alabama, USA. dohi@ri.imcj.go.jp | lld:pubmed |
pubmed-article:10982767 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10982767 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:10982767 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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