| pubmed-article:10970860 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10970860 | lifeskim:mentions | umls-concept:C0504064 | lld:lifeskim |
| pubmed-article:10970860 | lifeskim:mentions | umls-concept:C0001721 | lld:lifeskim |
| pubmed-article:10970860 | lifeskim:mentions | umls-concept:C0038952 | lld:lifeskim |
| pubmed-article:10970860 | lifeskim:mentions | umls-concept:C0001038 | lld:lifeskim |
| pubmed-article:10970860 | pubmed:issue | 17 | lld:pubmed |
| pubmed-article:10970860 | pubmed:dateCreated | 2000-10-24 | lld:pubmed |
| pubmed-article:10970860 | pubmed:abstractText | Acetylation of a transcription factor has recently been shown to play a significant role in gene regulation. Here we show that GATA-3 is acetylated in T cells and that a mutation introduced into amino acids 305-307 (KRR-GATA3) creates local hypoacetylation in GATA-3. Remarkably, KRR-GATA3 possesses the most potent suppressive effect when compared with other mutants that are disrupted in putative acetylation targets. Expressing this mutant in peripheral T cells results in defective T-cell homing to systemic lymphnodes, and prolonged T-cell survival after activation. These findings have significant implications in that the acetylation state of GATA-3 affects its physiological function in the immune system and, more importantly, provides evidence for the novel role of GATA-3 in T-cell survival and homing to secondary lymphoid organs. | lld:pubmed |
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| pubmed-article:10970860 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10970860 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10970860 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10970860 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10970860 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10970860 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10970860 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10970860 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:10970860 | pubmed:issn | 0261-4189 | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:SuzukiTT | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:HondaHH | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:HiraiHH | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:AsaiTT | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:MakiKK | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:NakamotoTT | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:OdaHH | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:MitaniKK | lld:pubmed |
| pubmed-article:10970860 | pubmed:author | pubmed-author:YamagataTT | lld:pubmed |
| pubmed-article:10970860 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10970860 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:10970860 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:10970860 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10970860 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10970860 | pubmed:pagination | 4676-87 | lld:pubmed |
| pubmed-article:10970860 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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