| pubmed-article:10948180 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0038172 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0014118 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0678226 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0699748 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0033453 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
| pubmed-article:10948180 | lifeskim:mentions | umls-concept:C0009082 | lld:lifeskim |
| pubmed-article:10948180 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:10948180 | pubmed:dateCreated | 2000-9-8 | lld:pubmed |
| pubmed-article:10948180 | pubmed:abstractText | Staphylococcus aureus Newman with an insertion mutation in clfB, the gene encoding clumping factor B, only marginally decreased infection rate (P>0.05) in rats with experimental endocarditis. In contrast, clfB complementation on a multicopy plasmid significantly increased infectivity (P<0.05) over the deleted mutants. Although clfB could affect endovascular infection, its importance in experimental endocarditis was limited. | lld:pubmed |
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| pubmed-article:10948180 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10948180 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10948180 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10948180 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10948180 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:10948180 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:FosterT JTJ | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:VaudauxPP | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:FrancioliPP | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:EntenzaJ MJM | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:Ni EidhinDD | lld:pubmed |
| pubmed-article:10948180 | pubmed:author | pubmed-author:MoreillonPP | lld:pubmed |
| pubmed-article:10948180 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10948180 | pubmed:volume | 68 | lld:pubmed |
| pubmed-article:10948180 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10948180 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10948180 | pubmed:pagination | 5443-6 | lld:pubmed |
| pubmed-article:10948180 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:10948180 | pubmed:meshHeading | pubmed-meshheading:10948180... | lld:pubmed |
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| pubmed-article:10948180 | pubmed:meshHeading | pubmed-meshheading:10948180... | lld:pubmed |
| pubmed-article:10948180 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10948180 | pubmed:articleTitle | Contribution of clumping factor B to pathogenesis of experimental endocarditis due to Staphylococcus aureus. | lld:pubmed |
| pubmed-article:10948180 | pubmed:affiliation | Division of Infectious Diseases, Department of Internal Medicine, Centre Hospitalier Universitaire Vaudois, 1011 Lausanne, Switzerland. | lld:pubmed |
| pubmed-article:10948180 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10948180 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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