| pubmed-article:10878028 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10878028 | lifeskim:mentions | umls-concept:C0038411 | lld:lifeskim |
| pubmed-article:10878028 | lifeskim:mentions | umls-concept:C0033105 | lld:lifeskim |
| pubmed-article:10878028 | lifeskim:mentions | umls-concept:C0370215 | lld:lifeskim |
| pubmed-article:10878028 | lifeskim:mentions | umls-concept:C0039260 | lld:lifeskim |
| pubmed-article:10878028 | lifeskim:mentions | umls-concept:C1710133 | lld:lifeskim |
| pubmed-article:10878028 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:10878028 | pubmed:dateCreated | 2000-9-1 | lld:pubmed |
| pubmed-article:10878028 | pubmed:abstractText | A total of 204 nonrepetitive isolates of group A streptococci (GAS), including 107 randomly collected between 1992 and 1995 and 66 and 31 consecutively collected in 1997 and 1998, respectively, from a university hospital in southern Taiwan were examined to determine the prevalence and mechanisms of erythromycin resistance among these isolates. Resistance to erythromycin was detected in 129 isolates (63.2%) by the agar dilution test. Of these, 42 isolates (32.6%) were assigned to the constitutive macrolide, lincosamide, and streptogramin B resistance (cMLS) phenotype, and all carried the ermB gene; 4 (3.1%) were assigned to the inducible MLS resistance (iMLS) phenotype, and all harbored the ermTR gene; and 83 (64.3%) were erythromycin resistant but susceptible to clindamycin (M phenotype), and all possessed the mefA gene. Distributed by years, the rates of erythromycin resistance and different phenotypes were 61.7% (53.0% cMLS, 6.1% iMLS, and 40.9% M phenotype) between 1992 and 1995, 62.1% (12.2% cMLS and 87.8% M phenotype) in 1997, and 71. 0% (9.1% cMLS and 90.9% M phenotype) in 1998. Pulsed-field gel electrophoresis showed that all but 2 cMLS isolates were clonal in origin, and 17 clones were detected among the M-phenotype isolates. These results indicate that the high incidence and increasing rate of erythromycin-resistant GAS in southern Taiwan are due to the prevalence of multiple M-phenotype clones and that clindamycin may be the drug of choice for the treatment of infections with GAS in penicillin-hypersensitive patients in this area. | lld:pubmed |
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| pubmed-article:10878028 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10878028 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10878028 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10878028 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:10878028 | pubmed:issn | 0095-1137 | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:LeeC TCT | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:HuangA HAH | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:YaoJ KJK | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:WuJ JJJ | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:WuH MHM | lld:pubmed |
| pubmed-article:10878028 | pubmed:author | pubmed-author:FuH MHM | lld:pubmed |
| pubmed-article:10878028 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10878028 | pubmed:volume | 38 | lld:pubmed |
| pubmed-article:10878028 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10878028 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10878028 | pubmed:pagination | 2475-9 | lld:pubmed |
| pubmed-article:10878028 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:10878028 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10878028 | pubmed:articleTitle | Prevalence of polyclonal mefA-containing isolates among erythromycin-resistant group A streptococci in Southern Taiwan. | lld:pubmed |
| pubmed-article:10878028 | pubmed:affiliation | Department of Pathology, National Cheng Kung University Medical Center, Tainan, Taiwan. | lld:pubmed |
| pubmed-article:10878028 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10878028 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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