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pubmed-article:10844628pubmed:abstractTextNephrotoxicity associated with cyclosporine A (CsA) administration is characterized by marked renal vasoconstriction, interstitial fibrosis, and arteriolar hypertrophy. While the molecular mechanisms of CsA toxicity are not well characterized, previous studies have demonstrated that altered arachidonic acid (AA) metabolism plays a role its pathogenesis. Using a rat renal transplant model, the purpose of this study was to examine the effects of CsA on the 5-lipoxygenase (5-LO) pathway of AA metabolism.lld:pubmed
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pubmed-article:10844628pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:10844628pubmed:articleTitleA role for leukotrienes in cyclosporine nephrotoxicity.lld:pubmed
pubmed-article:10844628pubmed:affiliationVA Medical Center, Durham, North Carolina 27705, USA.lld:pubmed
pubmed-article:10844628pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:10844628pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
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