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10821809
Source:
http://linkedlifedata.com/resource/pubmed/id/10821809
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pubmed-article:10821809
rdf:type
pubmed:Citation
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pubmed-article:10821809
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lifeskim:mentions
umls-concept:C0205191
lld:lifeskim
pubmed-article:10821809
pubmed:issue
20
lld:pubmed
pubmed-article:10821809
pubmed:dateCreated
2000-6-21
lld:pubmed
pubmed-article:10821809
pubmed:abstractText
The renin-angiotensin system may contribute to atherogenesis through the promotion of endothelial dysfunction. The present study was performed to determine whether angiotensin-1 (AT(1)) receptor inhibition improves endothelial dysfunction.
lld:pubmed
pubmed-article:10821809
pubmed:language
eng
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pubmed-article:10821809
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http://linkedlifedata.com/r...
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pubmed-article:10821809
pubmed:citationSubset
AIM
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pubmed:status
MEDLINE
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pubmed-article:10821809
pubmed:month
May
lld:pubmed
pubmed-article:10821809
pubmed:issn
1524-4539
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pubmed-article:10821809
pubmed:author
pubmed-author:PrasadAA
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pubmed-article:10821809
pubmed:author
pubmed-author:QuyyumiA AAA
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pubmed-article:10821809
pubmed:author
pubmed-author:PanzaJ AJA
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pubmed-article:10821809
pubmed:author
pubmed-author:EllahhamSS
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pubmed-article:10821809
pubmed:author
pubmed-author:SchenkeW HWH
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pubmed:author
pubmed-author:MincemoyerRR
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pubmed:author
pubmed-author:Tupas-HabibTT
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pubmed-article:10821809
pubmed:author
pubmed-author:WaclawinM AMA
lld:pubmed
pubmed-article:10821809
pubmed:issnType
Electronic
lld:pubmed
pubmed-article:10821809
pubmed:day
23
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pubmed-article:10821809
pubmed:volume
101
lld:pubmed
pubmed-article:10821809
pubmed:owner
NLM
lld:pubmed
pubmed-article:10821809
pubmed:authorsComplete
Y
lld:pubmed
pubmed-article:10821809
pubmed:pagination
2349-54
lld:pubmed
pubmed-article:10821809
pubmed:dateRevised
2010-11-18
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pubmed-article:10821809
pubmed:meshHeading
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lld:pubmed
pubmed-article:10821809
pubmed:year
2000
lld:pubmed
pubmed-article:10821809
pubmed:articleTitle
Acute and chronic angiotensin-1 receptor antagonism reverses endothelial dysfunction in atherosclerosis.
lld:pubmed
pubmed-article:10821809
pubmed:affiliation
Cardiology Branch, Office of Biostatistics Research, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD, USA.
lld:pubmed
pubmed-article:10821809
pubmed:publicationType
Journal Article
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pubmed-article:10821809
pubmed:publicationType
Clinical Trial
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pubmed-article:10821809
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