pubmed-article:10797167 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10797167 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
pubmed-article:10797167 | lifeskim:mentions | umls-concept:C0948008 | lld:lifeskim |
pubmed-article:10797167 | lifeskim:mentions | umls-concept:C0678226 | lld:lifeskim |
pubmed-article:10797167 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:10797167 | lifeskim:mentions | umls-concept:C0449560 | lld:lifeskim |
pubmed-article:10797167 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:10797167 | pubmed:dateCreated | 2000-5-31 | lld:pubmed |
pubmed-article:10797167 | pubmed:abstractText | Elevated plasma homocyst(e)ine may be a causal and modifiable risk factor for ischemic stroke, but the results of previous studies have been conflicting. One possible explanation is that homocyst(e)ine may only be associated with certain pathophysiological subtypes of ischemic stroke. | lld:pubmed |
pubmed-article:10797167 | pubmed:language | eng | lld:pubmed |
pubmed-article:10797167 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10797167 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:10797167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10797167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10797167 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10797167 | pubmed:month | May | lld:pubmed |
pubmed-article:10797167 | pubmed:issn | 0039-2499 | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:HanleyF JFJ | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:BalisS BSB | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:StaplesNN | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:AnandS SSS | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:EikelboomJ... | lld:pubmed |
pubmed-article:10797167 | pubmed:author | pubmed-author:LofthouseEE | lld:pubmed |
pubmed-article:10797167 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10797167 | pubmed:volume | 31 | lld:pubmed |
pubmed-article:10797167 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10797167 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10797167 | pubmed:pagination | 1069-75 | lld:pubmed |
pubmed-article:10797167 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
pubmed-article:10797167 | pubmed:meshHeading | pubmed-meshheading:10797167... | lld:pubmed |
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pubmed-article:10797167 | pubmed:meshHeading | pubmed-meshheading:10797167... | lld:pubmed |
pubmed-article:10797167 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:10797167 | pubmed:articleTitle | Association between high homocyst(e)ine and ischemic stroke due to large- and small-artery disease but not other etiologic subtypes of ischemic stroke. | lld:pubmed |
pubmed-article:10797167 | pubmed:affiliation | Preventive Cardiology and Therapeutics Program, McMaster University, Hamilton, Canada. | lld:pubmed |
pubmed-article:10797167 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10797167 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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