| pubmed-article:10770796 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0024264 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0299544 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0521447 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C1705630 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:10770796 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:10770796 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:10770796 | pubmed:dateCreated | 2000-5-9 | lld:pubmed |
| pubmed-article:10770796 | pubmed:abstractText | In lymphocytes, the Rel transcription factor is essential in establishing a pattern of gene expression that promotes cell proliferation, survival, and differentiation. Here we show that mitogen-induced expression of interferon (IFN) regulatory factor 4 (IRF-4), a lymphoid-specific member of the IFN family of transcription factors, is Rel dependent. Consistent with IRF-4 functioning as a repressor of IFN-induced gene expression, the absence of IRF-4 expression in c-rel(-/-) B cells coincided with a greater sensitivity of these cells to the antiproliferative activity of IFNs. In turn, enforced expression of an IRF-4 transgene restored IFN modulated c-rel(-/-) B cell proliferation to that of wild-type cells. This cross-regulation between two different signaling pathways represents a novel mechanism that Rel/nuclear factor kappaB can repress the transcription of IFN-regulated genes in a cell type-specific manner. | lld:pubmed |
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| pubmed-article:10770796 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10770796 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10770796 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10770796 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10770796 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10770796 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10770796 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10770796 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10770796 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:10770796 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:10770796 | pubmed:author | pubmed-author:GerondakisSS | lld:pubmed |
| pubmed-article:10770796 | pubmed:author | pubmed-author:GrumontR JRJ | lld:pubmed |
| pubmed-article:10770796 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10770796 | pubmed:day | 17 | lld:pubmed |
| pubmed-article:10770796 | pubmed:volume | 191 | lld:pubmed |
| pubmed-article:10770796 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10770796 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10770796 | pubmed:pagination | 1281-92 | lld:pubmed |
| pubmed-article:10770796 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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