10684298

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Subject	Predicate	Object	Context
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pubmed-article:10684298	lifeskim:mentions	umls-concept:C0206558	lld:lifeskim
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pubmed-article:10684298	lifeskim:mentions	umls-concept:C0085862	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1299583	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1704259	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1705987	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C0699789	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1947906	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1608386	lld:lifeskim
pubmed-article:10684298	lifeskim:mentions	umls-concept:C1549571	lld:lifeskim
pubmed-article:10684298	pubmed:issue	6	lld:pubmed
pubmed-article:10684298	pubmed:dateCreated	2000-4-3	lld:pubmed
pubmed-article:10684298	pubmed:abstractText	Cellular pre-mRNA splicing is inhibited by ICP27, a herpes simplex virus regulatory protein, resulting in the shutoff of host protein synthesis. Here we reveal that ICP27 also mediates the export of some virus RNAs via a Crm1-dependent pathway and present evidence that independent domains are required for these functions. Sorting of some viral mRNAs for nuclear export requires Crm1, while other virus mRNAs are exported via another pathway.	lld:pubmed
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