| pubmed-article:10671214 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0085732 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0598312 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1413236 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1317973 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0183683 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0344211 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1521721 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1515021 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0871161 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C1171411 | lld:lifeskim |
| pubmed-article:10671214 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:10671214 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:10671214 | pubmed:dateCreated | 2000-3-14 | lld:pubmed |
| pubmed-article:10671214 | pubmed:abstractText | Enrichment of a subset of CD4(+)CD45R0(+)CD7(-) T cells has been observed in HIV-infected individuals. We have investigated the ability of CD7(+) and CD7(-) T cells to support replication of HIV and show that virus replicates preferentially in CD7(+) cells. Several possible mechanisms that may underlie such differences in susceptibility to HIV were studied. Our data demonstrate that mitogen stimulation induces poor expression of CD25 and IL-2 in CD7(-) compared with CD7(+) cells. We also show that uninfected CD7(-) cells are more resistant to mitogen-induced apoptosis than CD7(+) cells. Our data support the view that the CD7(-) subset is inherently resistant to HIV replication and that this is due in part to reduced CD25 expression and IL-2 production. | lld:pubmed |
| pubmed-article:10671214 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10671214 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10671214 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10671214 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10671214 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10671214 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:10671214 | pubmed:issn | 0014-2980 | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:BeverleyP CPC | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:HicksRR | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:WallaceD LDL | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:DaviesD CDC | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:VyakarnamAA | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:MatearP MPM | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:LebosseCC | lld:pubmed |
| pubmed-article:10671214 | pubmed:author | pubmed-author:EyesonJJ | lld:pubmed |
| pubmed-article:10671214 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10671214 | pubmed:volume | 30 | lld:pubmed |
| pubmed-article:10671214 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10671214 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10671214 | pubmed:pagination | 577-85 | lld:pubmed |
| pubmed-article:10671214 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:meshHeading | pubmed-meshheading:10671214... | lld:pubmed |
| pubmed-article:10671214 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10671214 | pubmed:articleTitle | CD7 expression distinguishes subsets of CD4(+) T cells with distinct functional properties and ability to support replication of HIV-1. | lld:pubmed |
| pubmed-article:10671214 | pubmed:affiliation | Edward Jenner Institute for Vaccine Research, Compton, GB. diana.wallace@jenner.ac.uk | lld:pubmed |
| pubmed-article:10671214 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10671214 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10671214 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10671214 | lld:pubmed |
