| pubmed-article:10615996 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10615996 | lifeskim:mentions | umls-concept:C0003864 | lld:lifeskim |
| pubmed-article:10615996 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:10615996 | lifeskim:mentions | umls-concept:C0034790 | lld:lifeskim |
| pubmed-article:10615996 | lifeskim:mentions | umls-concept:C0023688 | lld:lifeskim |
| pubmed-article:10615996 | lifeskim:mentions | umls-concept:C0205359 | lld:lifeskim |
| pubmed-article:10615996 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:10615996 | pubmed:dateCreated | 2000-1-28 | lld:pubmed |
| pubmed-article:10615996 | pubmed:abstractText | Spontaneous arthritis in the KRN transgenic mouse model is due to the autoreactivity of the transgenic T cell receptor (TCR) against Ag7 major histocompatibility complex (MHC) molecules, which leads to strong but incomplete clonal deletion. We sought to determine whether other stimuli triggering this receptor might provoke arthritis, whether the apparently systemic reactivity might have some joint-preferential component explaining the paradoxical arthritic phenotype, and whether the transgenic receptor was the only one required or whether other TCRs might be ferried along in a leaky tolerance process. | lld:pubmed |
| pubmed-article:10615996 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10615996 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10615996 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:10615996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10615996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10615996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10615996 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10615996 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:10615996 | pubmed:issn | 0004-3591 | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:OokT HTH | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:BenoistCC | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:MathisDD | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:KorganowA SAS | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:KouskoffVV | lld:pubmed |
| pubmed-article:10615996 | pubmed:author | pubmed-author:MangialaioSS | lld:pubmed |
| pubmed-article:10615996 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10615996 | pubmed:volume | 42 | lld:pubmed |
| pubmed-article:10615996 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10615996 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10615996 | pubmed:pagination | 2517-23 | lld:pubmed |
| pubmed-article:10615996 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
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| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
| pubmed-article:10615996 | pubmed:meshHeading | pubmed-meshheading:10615996... | lld:pubmed |
| pubmed-article:10615996 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10615996 | pubmed:articleTitle | The arthritogenic T cell receptor and its ligand in a model of spontaneous arthritis. | lld:pubmed |
| pubmed-article:10615996 | pubmed:affiliation | Institut de Génétique et de Biologie Moléculaire et Cellulaire, Strasbourg, France. | lld:pubmed |
| pubmed-article:10615996 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10615996 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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