| pubmed-article:10599996 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0035243 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0035527 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0243127 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0040287 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0220927 | lld:lifeskim |
| pubmed-article:10599996 | lifeskim:mentions | umls-concept:C0443331 | lld:lifeskim |
| pubmed-article:10599996 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:10599996 | pubmed:dateCreated | 1999-12-29 | lld:pubmed |
| pubmed-article:10599996 | pubmed:abstractText | Studies in children and mice suggest that respiratory infections cause a mobilization of riboflavin from the tissues to the blood, resulting in increased urinary loss of this vitamin. To verify this observation, the tissue distribution and turnover of [3H]riboflavin were investigated in control and low-riboflavin-fed mice infected with Klebsiella pneumoniae. Infection significantly reduced [3H]riboflavin levels in the liver and kidney of low-riboflavin-fed mice and in the liver of control mice. Such changes were not observed in tissues such as muscle, small intestine, and brain. Urinary excretion of [3H]riboflavin increased significantly during the acute phase of infection and the biological half-life of [3H]riboflavin was shorter in the low-riboflavin-fed group. The results confirm that the mobilization of riboflavin from tissues to blood during infection results in a deterioration of riboflavin status. Thus, the study supports the hypothesis that respiratory infection is a nondietary factor contributing to the high prevalence of subclinical riboflavin deficiency in children of developing countries like India. | lld:pubmed |
| pubmed-article:10599996 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10599996 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10599996 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10599996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10599996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10599996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10599996 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10599996 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10599996 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:10599996 | pubmed:issn | 0026-0495 | lld:pubmed |
| pubmed-article:10599996 | pubmed:author | pubmed-author:LakshmiA VAV | lld:pubmed |
| pubmed-article:10599996 | pubmed:author | pubmed-author:BrijlalSS | lld:pubmed |
| pubmed-article:10599996 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10599996 | pubmed:volume | 48 | lld:pubmed |
| pubmed-article:10599996 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10599996 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10599996 | pubmed:pagination | 1608-11 | lld:pubmed |
| pubmed-article:10599996 | pubmed:dateRevised | 2005-11-17 | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
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| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
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| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:meshHeading | pubmed-meshheading:10599996... | lld:pubmed |
| pubmed-article:10599996 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10599996 | pubmed:articleTitle | Tissue distribution and turnover of [3H]riboflavin during respiratory infection in mice. | lld:pubmed |
| pubmed-article:10599996 | pubmed:affiliation | Department of Biochemistry, National Institute of Nutrition, Indian Council of Medical Research, Hyderabad. | lld:pubmed |
| pubmed-article:10599996 | pubmed:publicationType | Journal Article | lld:pubmed |
