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| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C1415288 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C2346484 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C1881189 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
| pubmed-article:10551584 | lifeskim:mentions | umls-concept:C0456148 | lld:lifeskim |
| pubmed-article:10551584 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:10551584 | pubmed:dateCreated | 1999-11-24 | lld:pubmed |
| pubmed-article:10551584 | pubmed:abstractText | Barbiturates enhance gamma-aminobutyric acid type A (GABA(A)) receptor function and also inhibit the alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) subtype of glutamate receptor. The relative contribution of these actions to the behavioral properties of barbiturates is not certain. Because AMPA receptor complexes that lack the GluR2 subunit are relatively insensitive to pentobarbital inhibition, GluR2 null mutant mice provide a novel tool to investigate the importance of AMPA receptor inhibition to the anesthetic effects of barbiturates. | lld:pubmed |
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| pubmed-article:10551584 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10551584 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10551584 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10551584 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:10551584 | pubmed:issn | 0003-3022 | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:NoeH NHN | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:MacDonaldJ... | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:RoderJJ | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:JinWW | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:XiongZZ | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:OrserB ABA | lld:pubmed |
| pubmed-article:10551584 | pubmed:author | pubmed-author:SonnerJJ | lld:pubmed |
| pubmed-article:10551584 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10551584 | pubmed:volume | 91 | lld:pubmed |
| pubmed-article:10551584 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10551584 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10551584 | pubmed:pagination | 1329-41 | lld:pubmed |
| pubmed-article:10551584 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:10551584 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10551584 | pubmed:articleTitle | Blockade of glutamate receptors and barbiturate anesthesia: increased sensitivity to pentobarbital-induced anesthesia despite reduced inhibition of AMPA receptors in GluR2 null mutant mice. | lld:pubmed |
| pubmed-article:10551584 | pubmed:affiliation | Department of Anaesthesia, University of Toronto, Ontario, Canada. | lld:pubmed |
| pubmed-article:10551584 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10551584 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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