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pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0004398 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0005558 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0868945 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0220825 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C1521733 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:10520942 | lifeskim:mentions | umls-concept:C0443172 | lld:lifeskim |
pubmed-article:10520942 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:10520942 | pubmed:dateCreated | 1999-10-28 | lld:pubmed |
pubmed-article:10520942 | pubmed:abstractText | To quantify the progression of senile plaques, neurofibrillary tangles, cerebral amyloid angiopathy, and microglial activation in the cortex and white matter of patients with Alzheimer disease evaluated at both biopsy and subsequent autopsy and correlate these changes with the progression of neurologic impairment. | lld:pubmed |
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pubmed-article:10520942 | pubmed:language | eng | lld:pubmed |
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pubmed-article:10520942 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:10520942 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10520942 | pubmed:month | Oct | lld:pubmed |
pubmed-article:10520942 | pubmed:issn | 0003-9942 | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:TomiyasuUU | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:CummingsJ LJL | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:VintersH VHV | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:SecorD LDL | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:RothF JFJJr | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:Di PatreP LPL | lld:pubmed |
pubmed-article:10520942 | pubmed:author | pubmed-author:VartavarianL... | lld:pubmed |
pubmed-article:10520942 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10520942 | pubmed:volume | 56 | lld:pubmed |
pubmed-article:10520942 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10520942 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10520942 | pubmed:pagination | 1254-61 | lld:pubmed |
pubmed-article:10520942 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:10520942 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:10520942 | pubmed:articleTitle | Progression of clinical deterioration and pathological changes in patients with Alzheimer disease evaluated at biopsy and autopsy. | lld:pubmed |
pubmed-article:10520942 | pubmed:affiliation | Department of Pathology and Laboratory Medicine, University of California, Los Angeles, Medical Center, 90095-1732, USA. dipatre@ucla.edu | lld:pubmed |
pubmed-article:10520942 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10520942 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:10520942 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:10520942 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:10520942 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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