| pubmed-article:10508678 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10508678 | lifeskim:mentions | umls-concept:C0012854 | lld:lifeskim |
| pubmed-article:10508678 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
| pubmed-article:10508678 | lifeskim:mentions | umls-concept:C0079866 | lld:lifeskim |
| pubmed-article:10508678 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:10508678 | pubmed:dateCreated | 1999-11-24 | lld:pubmed |
| pubmed-article:10508678 | pubmed:abstractText | Detailed analyses of mutational hotspots following DNA damage provide an understanding of oncogene activation and tumor suppressor gene inactivation, and hence provide an insight into the earliest steps in the induction of cancer. A mutational hotspot might be created by preferential lesion formation, decreased lesion repair, or increased misinsertion past the lesion during DNA replication. The respective contribution of these factors might be influenced by the DNA sequence context of the hotspot. | lld:pubmed |
| pubmed-article:10508678 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10508678 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10508678 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10508678 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10508678 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:10508678 | pubmed:issn | 1074-5521 | lld:pubmed |
| pubmed-article:10508678 | pubmed:author | pubmed-author:DelaneyJ CJC | lld:pubmed |
| pubmed-article:10508678 | pubmed:author | pubmed-author:EssigmannJ... | lld:pubmed |
| pubmed-article:10508678 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10508678 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:10508678 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10508678 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10508678 | pubmed:pagination | 743-53 | lld:pubmed |
| pubmed-article:10508678 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:meshHeading | pubmed-meshheading:10508678... | lld:pubmed |
| pubmed-article:10508678 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10508678 | pubmed:articleTitle | Context-dependent mutagenesis by DNA lesions. | lld:pubmed |
| pubmed-article:10508678 | pubmed:affiliation | Department of Chemistry, Division of Bioengineering and Environmental Health, Massachusetts Institute of Technology, Cambridge, MA 02139, USA. | lld:pubmed |
| pubmed-article:10508678 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10508678 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| entrez-gene:946710 | entrezgene:pubmed | pubmed-article:10508678 | lld:entrezgene |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10508678 | lld:pubmed |
