pubmed-article:10470199 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10470199 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:10470199 | lifeskim:mentions | umls-concept:C0007134 | lld:lifeskim |
pubmed-article:10470199 | lifeskim:mentions | umls-concept:C0005516 | lld:lifeskim |
pubmed-article:10470199 | lifeskim:mentions | umls-concept:C0034348 | lld:lifeskim |
pubmed-article:10470199 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
pubmed-article:10470199 | pubmed:issue | 4A | lld:pubmed |
pubmed-article:10470199 | pubmed:dateCreated | 1999-9-28 | lld:pubmed |
pubmed-article:10470199 | pubmed:abstractText | The evaluate a potential tumor marker for RCC. Tumor formation is generally linked to an expansion of glycolytic phosphometabolite pools and aerobic glycolyticflux rates. To achieve this, tumor cells generally overexpress a special glycolytic isoenzyme, termed pyruvate kinase type M2. To establish the expansion of phosphometabolite pools pyruvate kinase switches between a tetrameric form with high phosphoenol-pyruvate (PEP) affinity and a dimeric form with a lower PEP affinity. The dimeric form is predominant in all tumors that have been investigated and has been termed TuM2Pk. | lld:pubmed |
pubmed-article:10470199 | pubmed:language | eng | lld:pubmed |
pubmed-article:10470199 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10470199 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:10470199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10470199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10470199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10470199 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10470199 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10470199 | pubmed:issn | 0250-7005 | lld:pubmed |
pubmed-article:10470199 | pubmed:author | pubmed-author:BichlerK HKH | lld:pubmed |
pubmed-article:10470199 | pubmed:author | pubmed-author:FeinAA | lld:pubmed |
pubmed-article:10470199 | pubmed:author | pubmed-author:PetriEE | lld:pubmed |
pubmed-article:10470199 | pubmed:author | pubmed-author:WechselH WHW | lld:pubmed |
pubmed-article:10470199 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10470199 | pubmed:volume | 19 | lld:pubmed |
pubmed-article:10470199 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10470199 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10470199 | pubmed:pagination | 2583-90 | lld:pubmed |
pubmed-article:10470199 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
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pubmed-article:10470199 | pubmed:articleTitle | Marker for renal cell carcinoma (RCC): the dimeric form of pyruvate kinase type M2 (Tu M2-PK). | lld:pubmed |
pubmed-article:10470199 | pubmed:affiliation | Department of Urology, Eberhard-Karls-University, Tuebingen, Germany. | lld:pubmed |
pubmed-article:10470199 | pubmed:publicationType | Journal Article | lld:pubmed |
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