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pubmed-article:10468962pubmed:abstractTextPancreatic islet betacell tumours occur either sporadically or as part of inherited neoplastic syndromes, most commonly multiple endocrine neoplasia (MEN) type 1. Recently, a transgenic mouse model has been established in which the expression of the SV40 large T antigen was targeted to betacells by the rat insulin promoter, leading to the development of multiple pancreatic betacell tumours. In the advanced stages of tumour evolution, these tumours exhibited a high prevalence of loss of heterozygosity (LOH) on mouse chromosomes 9 and 16, at regions syntenic with regions 3q, 3p21, 6q12, 15q24 and 22q of the human genome.lld:pubmed
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pubmed-article:10468962pubmed:articleTitleFrequent loss of heterozygosity at chromosome 3p14.2-3p21 in human pancreatic islet cell tumours.lld:pubmed
pubmed-article:10468962pubmed:affiliationDivision of Endocrinology/Metabolism, Department of Medicine, University of Cincinnati Medical Center, Cincinnati, OH 45267-0547, USA.lld:pubmed
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