| pubmed-article:10466491 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C1510458 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C0004238 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C0013790 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C0205360 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C0442828 | lld:lifeskim |
| pubmed-article:10466491 | lifeskim:mentions | umls-concept:C1511559 | lld:lifeskim |
| pubmed-article:10466491 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:10466491 | pubmed:dateCreated | 1999-10-7 | lld:pubmed |
| pubmed-article:10466491 | pubmed:abstractText | Episodes of atrial fibrillation (AF) are known to cause both a rapid reduction in atrial refractoriness (atrial electrical remodeling) and a more delayed increase in AF stability in the chronic goat model. The aims of this study were to examine (1) the hypothesis that an AF-induced increase in AF stability might be due to a mechanism with a longer onset and offset than that of changes in refractoriness and (2) the possibility that repeated paroxysms of maintained AF might cause a cumulative increase in AF stability independent of changes in atrial refractoriness. | lld:pubmed |
| pubmed-article:10466491 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10466491 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10466491 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10466491 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10466491 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10466491 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:10466491 | pubmed:issn | 1045-3873 | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:MassGG | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:GarrattC JCJ | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:AllessieM AMA | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:KillianMM | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:DorlandRR | lld:pubmed |
| pubmed-article:10466491 | pubmed:author | pubmed-author:DuytschaeverM... | lld:pubmed |
| pubmed-article:10466491 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10466491 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:10466491 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10466491 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10466491 | pubmed:pagination | 1101-8 | lld:pubmed |
| pubmed-article:10466491 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:meshHeading | pubmed-meshheading:10466491... | lld:pubmed |
| pubmed-article:10466491 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10466491 | pubmed:articleTitle | Repetitive electrical remodeling by paroxysms of atrial fibrillation in the goat: no cumulative effect on inducibility or stability of atrial fibrillation. | lld:pubmed |
| pubmed-article:10466491 | pubmed:affiliation | Department of Physiology, Cardiovascular Research Institute Maastricht, University of Maastricht, The Netherlands. Clifford.Garratt@mh.cmht.nwest.nhs.uk | lld:pubmed |
| pubmed-article:10466491 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10466491 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:10466491 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10466491 | lld:pubmed |
