| pubmed-article:10441072 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10441072 | lifeskim:mentions | umls-concept:C0024660 | lld:lifeskim |
| pubmed-article:10441072 | lifeskim:mentions | umls-concept:C0007595 | lld:lifeskim |
| pubmed-article:10441072 | lifeskim:mentions | umls-concept:C1167093 | lld:lifeskim |
| pubmed-article:10441072 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:10441072 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:10441072 | pubmed:dateCreated | 1999-10-12 | lld:pubmed |
| pubmed-article:10441072 | pubmed:abstractText | The mammalian SWI/SNF complex is a chromatin remodelling complex that uses the energy of ATP hydrolysis to facilitate access of transcription factors to regulatory DNA sequences. This complex, that was initially described as a co-factor for nuclear receptors, has recently been associated with the control of cell growth. Two of the subunits known as BRG-1 and brm can associate with the Retinoblastoma tumour suppressor gene product and co-operate with this protein for repression of E2F activity. In addition, expression of brm is frequently down-regulated upon cellular transformation and re-introduction of this protein into fibroblasts transformed by activated ras induces partial reversion of the transformed phenotype. Finally, the hSNF5/INI1 gene, encoding another subunit of the SWI/SNF complex, is subject to bi-allelic mutations in rhabdoid tumours, a very aggressive form of paediatric cancers. These observations provide a novel link between malignant transformation and chromatin remodelling machineries. | lld:pubmed |
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| pubmed-article:10441072 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10441072 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10441072 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:10441072 | pubmed:issn | 1084-9521 | lld:pubmed |
| pubmed-article:10441072 | pubmed:author | pubmed-author:YanivMM | lld:pubmed |
| pubmed-article:10441072 | pubmed:author | pubmed-author:MuchardtCC | lld:pubmed |
| pubmed-article:10441072 | pubmed:copyrightInfo | Copyright 1999 Academic Press. | lld:pubmed |
| pubmed-article:10441072 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10441072 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:10441072 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10441072 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10441072 | pubmed:pagination | 189-95 | lld:pubmed |
| pubmed-article:10441072 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:10441072 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10441072 | pubmed:articleTitle | The mammalian SWI/SNF complex and the control of cell growth. | lld:pubmed |
| pubmed-article:10441072 | pubmed:affiliation | Unite@a3 des Virus Oncoge@a4nes, URA1644 du CNRS, De@a3partement des Biotechnologies, Institut Pasteur, 25 rue du Docteur Roux, Paris, Cedex 15, 75724, France. | lld:pubmed |
| pubmed-article:10441072 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10441072 | pubmed:publicationType | Review | lld:pubmed |
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