| pubmed-article:10373521 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10373521 | lifeskim:mentions | umls-concept:C0035679 | lld:lifeskim |
| pubmed-article:10373521 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
| pubmed-article:10373521 | lifeskim:mentions | umls-concept:C1423632 | lld:lifeskim |
| pubmed-article:10373521 | lifeskim:mentions | umls-concept:C0178555 | lld:lifeskim |
| pubmed-article:10373521 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:10373521 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:10373521 | pubmed:dateCreated | 1999-7-22 | lld:pubmed |
| pubmed-article:10373521 | pubmed:abstractText | Tat protein strongly activates transcription from the human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) by enhancing the elongation efficiency of RNA polymerase II complexes. Tat-mediated transcriptional activation requires cellular cofactors and specific cis-acting elements within the HIV-1 promoter, among them a functional TATA box. Here, we have investigated the mechanism by which one of these cofactors, termed CA150, regulates HIV-1 transcription in vivo. We present a series of functional assays that demonstrate that the regulation of the HIV-1 LTR by CA150 has the same functional requirements as the activation by Tat. We found that CA150 affects elongation of transcription complexes assembled on the HIV-1 promoter in a TATA-box-dependent manner. We discuss the data in terms of the involvement of CA150 in the regulation of Tat-activated HIV-1 gene expression. In addition, we also provide evidence suggesting a role for CA150 in the regulation of cellular transcriptional processes. | lld:pubmed |
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| pubmed-article:10373521 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10373521 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10373521 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10373521 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10373521 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:10373521 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:10373521 | pubmed:author | pubmed-author:Garcia-Blanco... | lld:pubmed |
| pubmed-article:10373521 | pubmed:author | pubmed-author:SuñéCC | lld:pubmed |
| pubmed-article:10373521 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10373521 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:10373521 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10373521 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10373521 | pubmed:pagination | 4719-28 | lld:pubmed |
| pubmed-article:10373521 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
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| pubmed-article:10373521 | pubmed:meshHeading | pubmed-meshheading:10373521... | lld:pubmed |
