| pubmed-article:10364917 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C0024141 | lld:lifeskim |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C0017431 | lld:lifeskim |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C0314657 | lld:lifeskim |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C1332828 | lld:lifeskim |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C1882932 | lld:lifeskim |
| pubmed-article:10364917 | lifeskim:mentions | umls-concept:C0332240 | lld:lifeskim |
| pubmed-article:10364917 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:10364917 | pubmed:dateCreated | 1999-6-21 | lld:pubmed |
| pubmed-article:10364917 | pubmed:abstractText | The aetiology of systemic lupus erythematosus (SLE) is still unknown. In several cases, however, chemicals or drugs were identified as aetiological agents and associations with certain phenotypes of drug metabolising enzymes have been reported. The purpose of this study was to discover if there is an association between CYP2C19 polymorphism and susceptibility to SLE. | lld:pubmed |
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| pubmed-article:10364917 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10364917 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10364917 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10364917 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10364917 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10364917 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10364917 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:10364917 | pubmed:issn | 0003-4967 | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:KayaalpS OSO | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:BrøsenKK | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:BozkurtAA | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:BasciN ENE | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:BathumLL | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:CalgüneriMM | lld:pubmed |
| pubmed-article:10364917 | pubmed:author | pubmed-author:KortunaySS | lld:pubmed |
| pubmed-article:10364917 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10364917 | pubmed:volume | 58 | lld:pubmed |
| pubmed-article:10364917 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10364917 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10364917 | pubmed:pagination | 182-5 | lld:pubmed |
| pubmed-article:10364917 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:10364917 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10364917 | pubmed:articleTitle | CYP2C19 genotype does not represent a genetic predisposition in idiopathic systemic lupus erythematosus. | lld:pubmed |
| pubmed-article:10364917 | pubmed:affiliation | Department of Pharmacology, Faculty of Medicine, Hacettepe University, Ankara, Turkey. | lld:pubmed |
| pubmed-article:10364917 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10364917 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10364917 | lld:pubmed |
