pubmed-article:10347347 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0027051 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0162577 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0456389 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0449295 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0175566 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0205225 | lld:lifeskim |
pubmed-article:10347347 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
pubmed-article:10347347 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:10347347 | pubmed:dateCreated | 1999-7-2 | lld:pubmed |
pubmed-article:10347347 | pubmed:abstractText | Several studies demonstrate a better outcome after primary angioplasty compared with thrombolysis. The mechanism is assumed to be a higher rate of open infarct-related vessels. | lld:pubmed |
pubmed-article:10347347 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10347347 | pubmed:language | eng | lld:pubmed |
pubmed-article:10347347 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10347347 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:10347347 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10347347 | pubmed:month | Jun | lld:pubmed |
pubmed-article:10347347 | pubmed:issn | 0002-8703 | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:de BoerM JMJ | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:SuryapranataH... | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:OttervangerJ... | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:LiemAA | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:ZijlstraFF | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:HoorntjeJ CJC | lld:pubmed |
pubmed-article:10347347 | pubmed:author | pubmed-author:van 't HofA... | lld:pubmed |
pubmed-article:10347347 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10347347 | pubmed:volume | 137 | lld:pubmed |
pubmed-article:10347347 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10347347 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10347347 | pubmed:pagination | 1169-72 | lld:pubmed |
pubmed-article:10347347 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:10347347 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:10347347 | pubmed:articleTitle | Limitation of myocardial infarct size after primary angioplasty: is a higher patency the only mechanism? | lld:pubmed |
pubmed-article:10347347 | pubmed:affiliation | Department of Cardiology, Hospital "De Weezenlanden", Zwolle, The Netherlands. | lld:pubmed |
pubmed-article:10347347 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10347347 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:10347347 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:10347347 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
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