| pubmed-article:10329985 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0026845 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C1995013 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0040715 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0015259 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C1522702 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0599718 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0599813 | lld:lifeskim |
| pubmed-article:10329985 | lifeskim:mentions | umls-concept:C0599893 | lld:lifeskim |
| pubmed-article:10329985 | pubmed:issue | 5 Pt 1 | lld:pubmed |
| pubmed-article:10329985 | pubmed:dateCreated | 1999-6-7 | lld:pubmed |
| pubmed-article:10329985 | pubmed:abstractText | It was recently found that the effect of an exercise-induced increase in muscle GLUT-4 on insulin-stimulated glucose transport is masked by a decreased responsiveness to insulin in glycogen-supercompensated muscle. We evaluated the role of hexosamines in this decrease in insulin responsiveness and found that UDP-N-acetyl hexosamine concentrations were not higher in glycogen-supercompensated muscles than in control muscles with a low glycogen content. We determined whether the smaller increase in glucose transport is due to translocation of fewer GLUT-4 to the cell surface with the 2-N-4-(1-azi-2,2,2-trifluroethyl)-benzoyl-1, 3-bis(D-mannose-4-yloxy)-2-propylamine (ATB-[2-3H]BMPA) photolabeling technique. The insulin-induced increase in GLUT-4 at the cell surface was no greater in glycogen-supercompensated exercised muscle than in muscles of sedentary controls and only 50% as great as in exercised muscles with a low glycogen content. We conclude that the decreased insulin responsiveness of glucose transport in glycogen-supercompensated muscle is not due to increased accumulation of hexosamine biosynthetic pathway end products and that the smaller increase in glucose transport is mediated by translocation of fewer GLUT-4 to the cell surface. | lld:pubmed |
| pubmed-article:10329985 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10329985 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10329985 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10329985 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10329985 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10329985 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10329985 | pubmed:month | May | lld:pubmed |
| pubmed-article:10329985 | pubmed:issn | 0002-9513 | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:HansenP APA | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:HolloszyJ OJO | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:NakataniAA | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:KIMS CSC | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:KawanakaKK | lld:pubmed |
| pubmed-article:10329985 | pubmed:author | pubmed-author:NolteL ALA | lld:pubmed |
| pubmed-article:10329985 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10329985 | pubmed:volume | 276 | lld:pubmed |
| pubmed-article:10329985 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10329985 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10329985 | pubmed:pagination | E907-12 | lld:pubmed |
| pubmed-article:10329985 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:10329985 | pubmed:meshHeading | pubmed-meshheading:10329985... | lld:pubmed |
| pubmed-article:10329985 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10329985 | pubmed:articleTitle | Decreased insulin-stimulated GLUT-4 translocation in glycogen-supercompensated muscles of exercised rats. | lld:pubmed |
| pubmed-article:10329985 | pubmed:affiliation | Department of Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA. | lld:pubmed |
| pubmed-article:10329985 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10329985 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:10329985 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:10329985 | lld:pubmed |
