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pubmed-article:10322353pubmed:issue5lld:pubmed
pubmed-article:10322353pubmed:dateCreated1999-12-22lld:pubmed
pubmed-article:10322353pubmed:abstractTextThe intraerythrocytic location of the malaria parasite necessitates modification of the host cell. These alterations are mediated either directly or indirectly by parasite proteins exported to specific compartments within the host cell. However, little is known about how the parasite specifically targets proteins to locations beyond its plasma membrane. Mark Wiser, Norbert Lanners and Richard Bafford here propose an alternative secretory pathway for the export of parasite proteins into the host erythrocyte. The first step of this pathway is probably an endoplasmic reticulum (ER)-like organelle that is distinct from the normal ER. Possible mechanisms of protein trafficking in the infected erythrocyte are also discussed. The proposed ER-like organelle and alternative secretory pathway raise many questions about the cell biology of protein export and trafficking in Plasmodium.lld:pubmed
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pubmed-article:10322353pubmed:monthMaylld:pubmed
pubmed-article:10322353pubmed:issn0169-4758lld:pubmed
pubmed-article:10322353pubmed:authorpubmed-author:LannersH NHNlld:pubmed
pubmed-article:10322353pubmed:authorpubmed-author:WiserM FMFlld:pubmed
pubmed-article:10322353pubmed:authorpubmed-author:BaffordR ARAlld:pubmed
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pubmed-article:10322353pubmed:volume15lld:pubmed
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pubmed-article:10322353pubmed:pagination194-8lld:pubmed
pubmed-article:10322353pubmed:dateRevised2005-11-16lld:pubmed
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pubmed-article:10322353pubmed:year1999lld:pubmed
pubmed-article:10322353pubmed:articleTitleExport of proteins via a novel secretory pathway.lld:pubmed
pubmed-article:10322353pubmed:affiliationDepartment of Tropical Medicine, Tulane University School of Public Health and Tropical Medicine, 1501 Canal Street, New Orleans, LA 70112, USA.wiser@mailhost.tcs.tulane.edulld:pubmed
pubmed-article:10322353pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:10322353pubmed:publicationTypeReviewlld:pubmed