| pubmed-article:10209007 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C1135918 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C1510411 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C0018270 | lld:lifeskim |
| pubmed-article:10209007 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
| pubmed-article:10209007 | pubmed:issue | 15 | lld:pubmed |
| pubmed-article:10209007 | pubmed:dateCreated | 1999-4-30 | lld:pubmed |
| pubmed-article:10209007 | pubmed:abstractText | It is postulated that vascular lesion formation and remodeling involves a balance between vascular cell death and cell proliferation. Transforming growth factor-beta1 (TGF-beta1) is a pleiotropic factor expressed within vascular cells that regulates cell growth in a tissue-specific manner. This study tested the hypothesis that the control of vascular cell apoptosis involves cell type-specific regulation by TGF-beta1. | lld:pubmed |
| pubmed-article:10209007 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10209007 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10209007 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10209007 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10209007 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10209007 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:10209007 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:10209007 | pubmed:author | pubmed-author:NaumovskiLL | lld:pubmed |
| pubmed-article:10209007 | pubmed:author | pubmed-author:GibbonsG HGH | lld:pubmed |
| pubmed-article:10209007 | pubmed:author | pubmed-author:PollmanM JMJ | lld:pubmed |
| pubmed-article:10209007 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:10209007 | pubmed:day | 20 | lld:pubmed |
| pubmed-article:10209007 | pubmed:volume | 99 | lld:pubmed |
| pubmed-article:10209007 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10209007 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10209007 | pubmed:pagination | 2019-26 | lld:pubmed |
| pubmed-article:10209007 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:10209007 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10209007 | pubmed:articleTitle | Vascular cell apoptosis: cell type-specific modulation by transforming growth factor-beta1 in endothelial cells versus smooth muscle cells. | lld:pubmed |
| pubmed-article:10209007 | pubmed:affiliation | Cardiovascular Research Laboratory, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115, USA. | lld:pubmed |
| pubmed-article:10209007 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10209007 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:10209007 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:10209007 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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