| pubmed-article:10190906 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0229664 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0023516 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0003320 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0019721 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C0019627 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C1145667 | lld:lifeskim |
| pubmed-article:10190906 | lifeskim:mentions | umls-concept:C1704241 | lld:lifeskim |
| pubmed-article:10190906 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:10190906 | pubmed:dateCreated | 1999-5-6 | lld:pubmed |
| pubmed-article:10190906 | pubmed:abstractText | The nonclassical MHC class I molecule human histocompatibility leukocyte antigen (HLA)-G is selectively expressed on fetal trophoblast tissue at the maternal-fetal interface in pregnancy. It has long been suggested that HLA-G may inhibit maternal natural killer (NK) cells through interaction with particular NK cell receptors (KIRs). To investigate interactions of HLA-G, we constructed phycoerythrin-labeled tetrameric complexes of HLA-G refolded with a self-peptide. These HLA-G tetramers failed to bind to NK cells and cells transfected with CD94/NKG2 and killer immunoglobulin-like NK receptors. In contrast, HLA-G tetramers did bind to peripheral blood monocytes, staining a CD16(+)CD14(mid) subset with greater intensity. On transfectants, HLA-G tetramers bound to inhibitory immunoglobulin-like transcript (ILT)2 and ILT4 receptors. However, staining in the presence of antibodies reactive with ILT receptors revealed that the interaction of HLA-G tetramers with blood monocytes was largely due to binding to ILT4. These results suggest that the primary role of HLA-G may be the modulation of myelomonocytic cell behavior in pregnancy. | lld:pubmed |
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| pubmed-article:10190906 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10190906 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10190906 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10190906 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10190906 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10190906 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:10190906 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:McMichaelA... | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:LanierL LLL | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:ChurakovaT... | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:AbramsJ SJS | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:ColonnaMM | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:EllisS ASA | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:AllanD SDS | lld:pubmed |
| pubmed-article:10190906 | pubmed:author | pubmed-author:BraudV MVM | lld:pubmed |
| pubmed-article:10190906 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10190906 | pubmed:day | 5 | lld:pubmed |
| pubmed-article:10190906 | pubmed:volume | 189 | lld:pubmed |
| pubmed-article:10190906 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10190906 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10190906 | pubmed:pagination | 1149-56 | lld:pubmed |
| pubmed-article:10190906 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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