| pubmed-article:10074209 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C0019682 | lld:lifeskim |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C0005953 | lld:lifeskim |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C0080194 | lld:lifeskim |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C0040034 | lld:lifeskim |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:10074209 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
| pubmed-article:10074209 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:10074209 | pubmed:dateCreated | 1999-5-6 | lld:pubmed |
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| pubmed-article:10074209 | pubmed:abstractText | We analyzed bone marrow and blood from human immunodeficiency virus type 1 (HIV-1)-infected individuals and described the HIV-1 quasispecies in these cellular compartments. HIV-1 isolates from the bone marrow of thrombocytopenic patients contained distinct amino acids in the V3 loop and infected T-cell lines, implicating this virus in the development of thrombocytopenia. | lld:pubmed |
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