<http://purl.uniprot.org/SHA-384/4E4C054BB08E9FBE0E513BC0B30AD4911BA47574C720FECAF45F874561E30423DE4D4E0C4D22E6E32C220AA52D079511> <http://www.w3.org/1999/02/22-rdf-syntax-ns#type> <http://purl.uniprot.org/core/Function_Annotation> .
<http://purl.uniprot.org/SHA-384/4E4C054BB08E9FBE0E513BC0B30AD4911BA47574C720FECAF45F874561E30423DE4D4E0C4D22E6E32C220AA52D079511> <http://www.w3.org/2000/01/rdf-schema#comment> "Alpha-conotoxins act on postsynaptic membranes, they bind to the nicotinic acetylcholine receptors (nAChR) and thus inhibit them. At 1 uM, the synthetic peptide reversibly blocks mammalian nAChR alpha-1/beta-1/gamma/delta and, to a lesser degree, alpha-6/alpha-3/beta-2, but has no effect on alpha-7. At 10 uM, it reversibly blocks nAChR alpha-3/beta-2, but has no effect on nAChR alpha-3/beta-4, alpha-4/beta-2, alpha-4/beta-4 or alpha-6/alpha-3/beta-4. Intramuscular injection of 10-20 ug of the synthetic peptide in goldfish causes death. Intravenous injection of the same doses in Kunming male mice causes unconsciousness. Does not inhibit potassium channels." .
<http://purl.uniprot.org/uniprot/C6ZJQ2> <http://purl.uniprot.org/core/annotation> <http://purl.uniprot.org/SHA-384/4E4C054BB08E9FBE0E513BC0B30AD4911BA47574C720FECAF45F874561E30423DE4D4E0C4D22E6E32C220AA52D079511> .
<http://linkedlifedata.com/resource/#_43365A4A5132008> <http://www.w3.org/1999/02/22-rdf-syntax-ns#subject> <http://purl.uniprot.org/SHA-384/4E4C054BB08E9FBE0E513BC0B30AD4911BA47574C720FECAF45F874561E30423DE4D4E0C4D22E6E32C220AA52D079511> .