| pubmed-article:3443963 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:3443963 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:3443963 | lifeskim:mentions | umls-concept:C0232804 | lld:lifeskim |
| pubmed-article:3443963 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
| pubmed-article:3443963 | pubmed:dateCreated | 1988-5-12 | lld:pubmed |
| pubmed-article:3443963 | pubmed:abstractText | 1. Standard renal clearance techniques were used to investigate the effects of chronic hyperprolactinaemia on kidney function in male, female and ovariectomized female rats. 2. All hyperprolactinaemic rats showed a significantly increased glomerular filtration rate (G.F.R.) compared to controls. Values were (microliter min-1) 2738 +/- 146 vs. 2299 +/- 99 for males (P less than 0.05), 2236 +/- 79 vs. 1865 +/- 74 for females (P less than 0.01) and 2200 +/- 76 vs. 1941 +/- 62 for ovariectomized females (P less than 0.05). 3. Hyperprolactinaemic rats in all groups also showed a significant increase in absolute tubular reabsorption of water, sodium and chloride compared to their respective controls. Increases here averaged 19%. 4. There was a significantly greater fractional tubular reabsorption of fluid and solutes in hyperprolactinaemic male rats compared to controls. Values were (%) 92.9 +/- 0.6 vs. 90.3 +/- 0.7 for water, 93.0 +/- 0.4 vs. 91.0 +/- 0.6 for sodium and 89.9 +/- 0.7 vs. 86.5 +/- 0.9 for chloride. In each case P less than 0.05. 5. These results imply an osmoregulatory role for prolactin which is not specific to pregnancy or related female reproductive states. | lld:pubmed |
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| pubmed-article:3443963 | pubmed:language | eng | lld:pubmed |
| pubmed-article:3443963 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:3443963 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:3443963 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:3443963 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:3443963 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:3443963 | pubmed:author | pubmed-author:GarlandH OHO | lld:pubmed |
| pubmed-article:3443963 | pubmed:author | pubmed-author:LewisA GAG | lld:pubmed |
| pubmed-article:3443963 | pubmed:author | pubmed-author:MilneC MCM | lld:pubmed |
| pubmed-article:3443963 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:3443963 | pubmed:volume | 394 | lld:pubmed |
| pubmed-article:3443963 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:3443963 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:3443963 | pubmed:pagination | 13-21 | lld:pubmed |
| pubmed-article:3443963 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:3443963 | pubmed:meshHeading | pubmed-meshheading:3443963-... | lld:pubmed |
| pubmed-article:3443963 | pubmed:year | 1987 | lld:pubmed |
| pubmed-article:3443963 | pubmed:articleTitle | Altered renal function in chronically hyperprolactinaemic rats. | lld:pubmed |
| pubmed-article:3443963 | pubmed:affiliation | Department of Physiological Sciences, University of Manchester. | lld:pubmed |
| pubmed-article:3443963 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:3443963 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
