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pubmed-article:20186099pubmed:abstractTextA number of factors contribute to diabetes-associated vascular dysfunction. In the present study, we tested whether exposure to advanced glycation end-products (AGEs) impairs vascular reactivity independently of hyperglycemia and examined the potential mechanisms responsible for diabetes and AGE-associated vascular dysfunction.lld:pubmed
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pubmed-article:20186099pubmed:articleTitleAdvanced glycation end-products induce vascular dysfunction via resistance to nitric oxide and suppression of endothelial nitric oxide synthase.lld:pubmed
pubmed-article:20186099pubmed:affiliationJDRF Einstein Centre for Diabetes Complications, Melbourne, Australia.lld:pubmed
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