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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2010-7-16
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pubmed:abstractText |
A number of factors contribute to diabetes-associated vascular dysfunction. In the present study, we tested whether exposure to advanced glycation end-products (AGEs) impairs vascular reactivity independently of hyperglycemia and examined the potential mechanisms responsible for diabetes and AGE-associated vascular dysfunction.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1473-5598
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pubmed:author |
pubmed-author:BrasacchioDaniellaD,
pubmed-author:Chin-DustingJayeJ,
pubmed-author:CooperMark EME,
pubmed-author:CoughlanMelinda TMT,
pubmed-author:ForbesJosephine MJM,
pubmed-author:HarrisEmmaE,
pubmed-author:KayeDavidD,
pubmed-author:PaavonenKarriK,
pubmed-author:Soro-PaavonenAinoA,
pubmed-author:ThomasMerlin CMC,
pubmed-author:TongDavid C KDC,
pubmed-author:VenardosKylieK,
pubmed-author:ZhangWei-ZengWZ
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pubmed:issnType |
Electronic
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pubmed:volume |
28
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
780-8
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pubmed:meshHeading |
pubmed-meshheading:20186099-Animals,
pubmed-meshheading:20186099-Aorta, Thoracic,
pubmed-meshheading:20186099-Arginine,
pubmed-meshheading:20186099-Cattle,
pubmed-meshheading:20186099-Cells, Cultured,
pubmed-meshheading:20186099-Diabetes Mellitus,
pubmed-meshheading:20186099-Diabetes Mellitus, Experimental,
pubmed-meshheading:20186099-Diabetes Mellitus, Type 2,
pubmed-meshheading:20186099-Endothelial Cells,
pubmed-meshheading:20186099-Glycosylation End Products, Advanced,
pubmed-meshheading:20186099-Hypoglycemic Agents,
pubmed-meshheading:20186099-Male,
pubmed-meshheading:20186099-Nitric Oxide,
pubmed-meshheading:20186099-Nitric Oxide Synthase Type III,
pubmed-meshheading:20186099-Rats,
pubmed-meshheading:20186099-Rats, Sprague-Dawley,
pubmed-meshheading:20186099-Vascular Diseases,
pubmed-meshheading:20186099-Vasodilation,
pubmed-meshheading:20186099-Vasodilator Agents
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pubmed:year |
2010
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pubmed:articleTitle |
Advanced glycation end-products induce vascular dysfunction via resistance to nitric oxide and suppression of endothelial nitric oxide synthase.
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pubmed:affiliation |
JDRF Einstein Centre for Diabetes Complications, Melbourne, Australia.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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