rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
12
|
pubmed:dateCreated |
2008-12-23
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pubmed:abstractText |
Lung transplantation is a well accepted therapy for end-stage lung disease, despite high mortality rates. Mortality after transplantation is mainly caused by allograft failure in the first years after transplantation. Mannose binding lectin (MBL), a recognition molecule of innate immunity, has been associated with transplant outcome in other solid organ transplantation. In this study, the effect of donor- and recipient-MBL genotype on lung transplant outcome was investigated.
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:month |
Dec
|
pubmed:issn |
1534-6080
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:day |
27
|
pubmed:volume |
86
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1857-63
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pubmed:meshHeading |
pubmed-meshheading:19104434-Adult,
pubmed-meshheading:19104434-Bronchiolitis Obliterans,
pubmed-meshheading:19104434-DNA,
pubmed-meshheading:19104434-Female,
pubmed-meshheading:19104434-Genotype,
pubmed-meshheading:19104434-Graft Survival,
pubmed-meshheading:19104434-Humans,
pubmed-meshheading:19104434-Lung Diseases,
pubmed-meshheading:19104434-Lung Transplantation,
pubmed-meshheading:19104434-Male,
pubmed-meshheading:19104434-Mannose-Binding Lectins,
pubmed-meshheading:19104434-Middle Aged,
pubmed-meshheading:19104434-Polymorphism, Genetic,
pubmed-meshheading:19104434-Polymorphism, Single Nucleotide,
pubmed-meshheading:19104434-Promoter Regions, Genetic,
pubmed-meshheading:19104434-Retrospective Studies,
pubmed-meshheading:19104434-Young Adult
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pubmed:year |
2008
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pubmed:articleTitle |
Association between donor MBL promoter haplotype and graft survival and the development of BOS after lung transplantation.
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pubmed:affiliation |
Department of Internal Medicine, University Medical Center Groningen, Groningen, The Netherlands. j.m.munster@int.umcg.nl
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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