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| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0345904 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0019647 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C1367731 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0025723 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
| pubmed-article:19041150 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:19041150 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:19041150 | pubmed:dateCreated | 2009-1-16 | lld:pubmed |
| pubmed-article:19041150 | pubmed:abstractText | Aberrant c-Jun N-terminal kinase (JNK) activation has been linked to hepatocellular carcinoma (HCC) in mouse models. It remains unclear whether JNK activation plays an important role in human HCC and, if so, how JNK signaling contributes to the initiation or progression of HCC. | lld:pubmed |
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| pubmed-article:19041150 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19041150 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19041150 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19041150 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:19041150 | pubmed:issn | 0168-8278 | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ChenJianguoJ | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ChenFeiF | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ShiXianglinX | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:CastranovaVin... | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ZhangYadongY | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ZhaoHongwenH | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:BhatiaDeepakD | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:ChangQingshan... | lld:pubmed |
| pubmed-article:19041150 | pubmed:author | pubmed-author:BeezholdKevin... | lld:pubmed |
| pubmed-article:19041150 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:19041150 | pubmed:volume | 50 | lld:pubmed |
| pubmed-article:19041150 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19041150 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19041150 | pubmed:pagination | 323-33 | lld:pubmed |
| pubmed-article:19041150 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:19041150 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19041150 | pubmed:articleTitle | Sustained JNK1 activation is associated with altered histone H3 methylations in human liver cancer. | lld:pubmed |
| pubmed-article:19041150 | pubmed:affiliation | Graduate Center for Toxicology, University of Kentucky, Lexington, KY 40536, USA. | lld:pubmed |
| pubmed-article:19041150 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19041150 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| pubmed-article:19041150 | pubmed:publicationType | Research Support, N.I.H., Intramural | lld:pubmed |
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